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Hence, it's conceivable that the JIP1/3???ASK1???MKK4 signaling module may possibly serve as a conduit to activate JNK when endosomal trafficking is disrupted and glucose concentrations drop in the course of the early levels of methuosis. Our finding that MKK4 was robustly activated through the methuosis-inducing IPPs, MOMIPP and 2q, and less so fr